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@article{mclaughlin_child_2015,
title = {Child maltreatment and neural systems underlying emotion regulation},
volume = {54},
issn = {1527-5418},
doi = {10.1016/j.jaac.2015.06.010},
abstract = {OBJECTIVE: The strong associations between child maltreatment and psychopathology have generated interest in identifying neurodevelopmental processes that are disrupted following maltreatment. Previous research has focused largely on neural response to negative facial emotion. We determined whether child maltreatment was associated with neural responses during passive viewing of negative and positive emotional stimuli and effortful attempts to regulate emotional responses.
METHOD: A total of 42 adolescents aged 13 to 19 years, half with exposure to physical and/or sexual abuse, participated. Blood oxygen level-dependent (BOLD) response was measured during passive viewing of negative and positive emotional stimuli and attempts to modulate emotional responses using cognitive reappraisal.
RESULTS: Maltreated adolescents exhibited heightened response in multiple nodes of the salience network, including amygdala, putamen, and anterior insula, to negative relative to neutral stimuli. During attempts to decrease responses to negative stimuli relative to passive viewing, maltreatment was associated with greater recruitment of superior frontal gyrus, dorsal anterior cingulate cortex, and frontal pole; adolescents with and without maltreatment down-regulated amygdala response to a similar degree. No associations were observed between maltreatment and neural response to positive emotional stimuli during passive viewing or effortful regulation.
CONCLUSION: Child maltreatment heightens the salience of negative emotional stimuli. Although maltreated adolescents modulate amygdala responses to negative cues to a degree similar to that of non-maltreated youths, they use regions involved in effortful control to a greater degree to do so, potentially because greater effort is required to modulate heightened amygdala responses. These findings are promising, given the centrality of cognitive restructuring in trauma-focused treatments for children.},
language = {eng},
number = {9},
journal = {Journal of the American Academy of Child and Adolescent Psychiatry},
author = {McLaughlin, Katie A. and Peverill, Matthew and Gold, Andrea L. and Alves, Sonia and Sheridan, Margaret A.},
month = sep,
year = {2015},
pmid = {26299297},
pmcid = {PMC4548288},
pages = {753--762},
file = {McLaughlin et al. - 2015 - Child Maltreatment and Neural Systems Underlying E.pdf:C\:\\Users\\Matt Peverill\\Zotero\\storage\\T93ANPFT\\McLaughlin et al. - 2015 - Child Maltreatment and Neural Systems Underlying E.pdf:application/pdf;NIHMS705725-supplement.pdf:C\:\\Users\\Matt Peverill\\Zotero\\storage\\A84BVVT9\\NIHMS705725-supplement.pdf:application/pdf}
}
@article{askren_using_2016,
title = {Using {Make} for reproducible and parallel neuroimaging workflow and quality-assurance},
volume = {10},
issn = {1662-5196},
doi = {10.3389/fninf.2016.00002},
abstract = {The contribution of this paper is to describe how we can program neuroimaging workflow using Make, a software development tool designed for describing how to build executables from source files. A makefile (or a file of instructions for Make) consists of a set of rules that create or update target files if they have not been modified since their dependencies were last modified. These rules are processed to create a directed acyclic dependency graph that allows multiple entry points from which to execute the workflow. We show that using Make we can achieve many of the features of more sophisticated neuroimaging pipeline systems, including reproducibility, parallelization, fault tolerance, and quality assurance reports. We suggest that Make permits a large step toward these features with only a modest increase in programming demands over shell scripts. This approach reduces the technical skill and time required to write, debug, and maintain neuroimaging workflows in a dynamic environment, where pipelines are often modified to accommodate new best practices or to study the effect of alternative preprocessing steps, and where the underlying packages change frequently. This paper has a comprehensive accompanying manual with lab practicals and examples (see Supplemental Materials) and all data, scripts, and makefiles necessary to run the practicals and examples are available in the "makepipelines" project at NITRC.},
language = {eng},
journal = {Frontiers in Neuroinformatics},
author = {Askren, Mary K. and McAllister-Day, Trevor K. and Koh, Natalie and Mestre, Zoé and Dines, Jennifer N. and Korman, Benjamin A. and Melhorn, Susan J. and Peterson, Daniel J. and Peverill, Matthew and Qin, Xiaoyan and Rane, Swati D. and Reilly, Melissa A. and Reiter, Maya A. and Sambrook, Kelly A. and Woelfer, Karl A. and Grabowski, Thomas J. and Madhyastha, Tara M.},
year = {2016},
pmid = {26869916},
pmcid = {PMC4735413},
keywords = {neuroimaging pipelines, quality assurance, reproducibility, workflow},
pages = {2}
}
@article{mclaughlin_maltreatment_2016,
title = {Maltreatment exposure, brain structure, and fear conditioning in children and adolescents},
issn = {1740-634X},
doi = {10.1038/npp.2015.365},
abstract = {Alterations in learning processes and the neural circuitry that supports fear conditioning and extinction represent mechanisms through which trauma exposure might influence risk for psychopathology. Few studies examine how trauma or neural structure relates to fear conditioning in children. Children (n=94) aged 6-18 years, 40.4\% (n=38) with exposure to maltreatment (physical abuse, sexual abuse, or domestic violence), completed a fear conditioning paradigm utilizing blue and yellow bells as conditioned stimuli (CS+/CS-) and an aversive alarm noise as the unconditioned stimulus. Skin conductance responses (SCR) and self-reported fear were acquired. Magnetic resonance imaging data were acquired from 60 children. Children without maltreatment exposure exhibited strong differential conditioning to the CS+ vs CS-, based on SCR and self-reported fear. In contrast, maltreated children exhibited blunted SCR to the CS+ and failed to exhibit differential SCR to the CS+ vs CS- during early conditioning. Amygdala and hippocampal volume were reduced among children with maltreatment exposure and were negatively associated with SCR to the CS+ during early conditioning in the total sample, although these associations were negative only among non-maltreated children and were positive among maltreated children. The association of maltreatment with externalizing psychopathology was mediated by this perturbed pattern of fear conditioning. Child maltreatment is associated with failure to discriminate between threat and safety cues during fear conditioning in children. Poor threat-safety discrimination might reflect either enhanced fear generalization or a deficit in associative learning, which may in turn represent a central mechanism underlying the development of maltreatment-related externalizing psychopathology in children.Neuropsychopharmacology advance online publication, 13 January 2016; doi:10.1038/npp.2015.365.},
language = {ENG},
journal = {Neuropsychopharmacology},
author = {McLaughlin, Katie A. and Sheridan, Margaret A. and Gold, Andrea L. and Duys, Andrea and Lambert, Hilary K. and Peverill, Matthew and Heleniak, Charlotte and Shechner, Tomer and Wojcieszak, Zuzanna and Pine, Daniel S.},
month = jul,
year = {2016},
pmid = {26677946},
file = {McLaughlin et al. - 2015 - Maltreatment exposure, brain structure, and fear c.pdf:C\:\\Users\\Matt Peverill\\Zotero\\storage\\UAFNDM58\\McLaughlin et al. - 2015 - Maltreatment exposure, brain structure, and fear c.pdf:application/pdf}
}
@article{peverill_working_2016,
title = {Working memory filtering continues to develop into late adolescence},
issn = {1878-9293},
url = {http://www.sciencedirect.com/science/article/pii/S1878929315300177},
doi = {10.1016/j.dcn.2016.02.004},
abstract = {While most measures of working memory (WM) performance have been shown to plateau by mid-adolescence and developmental changes in fronto-parietal regions supporting WM encoding and maintenance have been well characterized, little is known about developmental variation in WM filtering. We investigated the possibility that the neural underpinnings of filtering in WM reach maturity later in life than WM function without filtering. Using a cued WM filtering task (McNab and Klingberg, 2008), we investigated neural activity during WM filtering in a sample of 64 adults and adolescents. Regardless of age, increases in WM activity with load were concentrated in the expected fronto-parietal network. For adults, but not adolescents, recruitment of the basal ganglia during presentation of a filtering cue was associated with neural and behavioral indices of successful filtering, suggesting that WM filtering and related basal ganglia function may still be maturing throughout adolescence and into adulthood.},
urldate = {2016-03-30},
journal = {Developmental Cognitive Neuroscience},
author = {Peverill, Matthew and McLaughlin, Katie A. and Finn, Amy S. and Sheridan, Margaret A.},
year = {2016},
keywords = {ADOLESCENCE, basal ganglia, Working memory, Working memory filtering},
file = {ScienceDirect Full Text PDF:C\:\\Users\\Matt Peverill\\Zotero\\storage\\2M2GIGMS\\Peverill et al. - Working memory filtering continues to develop into.pdf:application/pdf;ScienceDirect Snapshot:C\:\\Users\\Matt Peverill\\Zotero\\storage\\5SAMTREI\\S1878929315300177.html:text/html}
}
@article{gold_childhood_2016,
title = {Childhood abuse and reduced cortical thickness in brain regions involved in emotional processing},
volume = {57},
issn = {1469-7610},
url = {http://onlinelibrary.wiley.com/doi/10.1111/jcpp.12630/abstract},
doi = {10.1111/jcpp.12630},
abstract = {Background
Alterations in gray matter development represent a potential pathway through which childhood abuse is associated with psychopathology. Several prior studies find reduced volume and thickness of prefrontal (PFC) and temporal cortex regions in abused compared with nonabused adolescents, although most prior research is based on adults and volume-based measures. This study tests the hypothesis that child abuse, independent of parental education, predicts reduced cortical thickness in prefrontal and temporal cortices as well as reduced gray mater volume (GMV) in subcortical regions during adolescence.
Methods
Structural MRI scans were obtained from 21 adolescents exposed to physical and/or sexual abuse and 37 nonabused adolescents (ages 13–20). Abuse was operationalized using dichotomous and continuous measures. We examined associations between abuse and brain structure in several a priori-defined regions, controlling for parental education, age, sex, race, and total brain volume for subcortical GMV. Significance was evaluated at p {\textless} .05 with a false discovery rate correction.
Results
Child abuse exposure and severity were associated with reduced thickness in ventromedial prefrontal cortex (PFC), right lateral orbitofrontal cortex, right inferior frontal gyrus, bilateral parahippocampal gyrus (PHG), left temporal pole, and bilateral inferior, right middle, and right superior temporal gyri. Neither abuse measure predicted cortical surface area or subcortical GMV. Bilateral PHG thickness was inversely related to externalizing symptoms.
Conclusions
Child abuse, an experience characterized by a high degree of threat, is associated with reduced cortical thickness in ventromedial and ventrolateral PFC and medial and lateral temporal cortex in adolescence. Reduced PHG thickness may be a mediator linking abuse with externalizing psychopathology, although prospective research is needed to evaluate this possibility.},
language = {en},
number = {10},
urldate = {2016-09-22},
journal = {Journal of Child Psychology and Psychiatry},
author = {Gold, Andrea L. and Sheridan, Margaret A. and Peverill, Matthew and Busso, Daniel S. and Lambert, Hilary K. and Alves, Sonia and Pine, Daniel S. and McLaughlin, Katie A.},
month = oct,
year = {2016},
keywords = {Abuse, childhood adversity, cortical thickness, temporal cortex, ventromedial prefrontal cortex},
pages = {1154--1164},
file = {Full Text PDF:C\:\\Users\\Matt Peverill\\Zotero\\storage\\RM4RUV2E\\Gold et al. - 2016 - Childhood abuse and reduced cortical thickness in .pdf:application/pdf;Snapshot:C\:\\Users\\Matt Peverill\\Zotero\\storage\\7E77CBSV\\abstract.html:text/html}
}
@article{dennison_neurobehavioral_2016,
title = {Neurobehavioral markers of resilience to depression amongst adolescents exposed to child abuse},
volume = {125},
issn = {1939-1846},
doi = {10.1037/abn0000215},
abstract = {Childhood maltreatment is strongly associated with depression, which is characterized by reduced reactivity to reward. Identifying factors that mitigate risk for depression in maltreated children is important for understanding etiological links between maltreatment and depression as well as improving early intervention and prevention. We examine whether high reward reactivity at behavioral and neurobiological levels is a marker of resilience to depressive symptomology in adolescence following childhood maltreatment. A sample of 59 adolescents (21 with a history of maltreatment; Mean Age = 16.95 years, SD = 1.44) completed an fMRI task involving passive viewing of emotional stimuli. BOLD signal changes to positive relative to neutral images were extracted in basal ganglia regions of interest. Participants also completed a behavioral reward-processing task outside the scanner. Depression symptoms were assessed at the time of the MRI and again 2 years later. Greater reward reactivity across behavioral and neurobiological measures moderated the association of maltreatment with baseline depression. Specifically, faster reaction time (RT) to cues paired with monetary reward relative to those unpaired with reward and greater BOLD signal in the left pallidum was associated with lower depression symptoms in maltreated youth. Longitudinally, greater BOLD signal in the left putamen moderated change in depression scores over time, such that higher levels of reward response were associated with lower increases in depression over time among maltreated youths. Reactivity to monetary reward and positive social images, at both behavioral and neurobiological levels, is a potential marker of resilience to depression among adolescents exposed to maltreatment. These findings add to a growing body of work highlighting individual differences in reactivity to reward as a core neurodevelopmental mechanism in the etiology of depression. (PsycINFO Database Record},
language = {eng},
number = {8},
journal = {Journal of Abnormal Psychology},
author = {Dennison, Meg J. and Sheridan, Margaret A. and Busso, Daniel S. and Jenness, Jessica L. and Peverill, Matthew and Rosen, Maya L. and McLaughlin, Katie A.},
year = {2016},
pmid = {27819477},
pmcid = {PMC5119749},
pages = {1201--1212}
}
@article{busso_child_2017,
title = {Child {Abuse}, {Neural} {Structure}, and {Adolescent} {Psychopathology}: {A} {Longitudinal} {Study}},
volume = {56},
issn = {0890-8567, 1527-5418},
shorttitle = {Child {Abuse}, {Neural} {Structure}, and {Adolescent} {Psychopathology}},
url = {http://jaacap.com/article/S0890-8567(17)30026-6/abstract},
doi = {10.1016/j.jaac.2017.01.013},
language = {English},
number = {4},
urldate = {2017-04-03},
journal = {Journal of the American Academy of Child \& Adolescent Psychiatry},
author = {Busso, Daniel S. and McLaughlin, Katie A. and Brueck, Stephanie and Peverill, Matthew and Gold, Andrea L. and Sheridan, Margaret A.},
month = apr,
year = {2017},
pmid = {28335876},
keywords = {Child Abuse, neural structure, parahippocampal gyrus, Psychopathology, trauma},
pages = {321--328.e1},
file = {Busso et al. - 2017 - Child Abuse, Neural Structure, and Adolescent Psyc.pdf:C\:\\Users\\Matt Peverill\\Zotero\\storage\\62JM4J3R\\Busso et al. - 2017 - Child Abuse, Neural Structure, and Adolescent Psyc.pdf:application/pdf;Snapshot:C\:\\Users\\Matt Peverill\\Zotero\\storage\\ZZCQPAFK\\abstract.html:text/html}
}
@article{sun_structural_2018,
title = {Structural covariance network centrality in maltreated youth with posttraumatic stress disorder},
volume = {98},
issn = {0022-3956},
url = {http://www.sciencedirect.com/science/article/pii/S0022395617301267},
doi = {10.1016/j.jpsychires.2017.12.015},
abstract = {Childhood maltreatment is associated with posttraumatic stress disorder (PTSD) and elevated rates of adolescent and adult psychopathology including major depression, bipolar disorder, substance use disorders, and other medical comorbidities. Gray matter volume changes have been found in maltreated youth with (versus without) PTSD. However, little is known about the alterations of brain structural covariance network topology derived from cortical thickness in maltreated youth with PTSD. High-resolution T1-weighted magnetic resonance imaging scans were from demographically matched maltreated youth with PTSD (N = 24), without PTSD (N = 64), and non-maltreated healthy controls (n = 67). Cortical thickness data from 148 cortical regions was entered into interregional partial correlation analyses across participants. The supra-threshold correlations constituted connections in a structural brain network derived from four types of centrality measures (degree, betweenness, closeness, and eigenvector) estimated network topology and the importance of nodes. Between-group differences were determined by permutation testing. Maltreated youth with PTSD exhibited larger centrality in left anterior cingulate cortex than the other two groups, suggesting cortical network topology specific to maltreated youth with PTSD. Moreover, maltreated youth with versus without PTSD showed smaller centrality in right orbitofrontal cortex, suggesting that this may represent a vulnerability factor to PTSD following maltreatment. Longitudinal follow-up of the present results will help characterize the role that altered centrality plays in vulnerability and resilience to PTSD following childhood maltreatment.},
journal = {Journal of Psychiatric Research},
author = {Sun, Delin and Peverill, Matthew R. and Swanson, Chelsea S. and McLaughlin, Katie A. and Morey, Rajendra A.},
month = mar,
year = {2018},
keywords = {anterior cingulate cortex, Centrality, Childhood maltreatment, cortical thickness, posttraumatic stress disorder, Structural covariance network},
pages = {70--77},
file = {ScienceDirect Full Text PDF:C\:\\Users\\Matt Peverill\\Zotero\\storage\\ZU4GC7DA\\Sun et al. - 2018 - Structural covariance network centrality in maltre.pdf:application/pdf;ScienceDirect Snapshot:C\:\\Users\\Matt Peverill\\Zotero\\storage\\VR98BCGJ\\S0022395617301267.html:text/html}
}
@article{miller_neural_2017,
title = {Neural {Correlates} of {Emotion} {Regulation} and {Adolescent} {Suicidal} {Ideation}},
issn = {2451-9022},
url = {http://www.sciencedirect.com/science/article/pii/S2451902217301489},
doi = {10.1016/j.bpsc.2017.08.008},
abstract = {Research on the neural correlates associated with risk for suicidal ideation (SI) has been limited, particularly in one increasingly at-risk group—adolescents. Previous research with adolescents indicates that poor emotion regulation skills are linked with SI, but these studies have not previously examined neural activation in service of emotion regulation between those with and without SI histories. Here we examine whether SI is associated with neural responses during an emotion regulation functional magnetic resonance imaging task in a group of adolescents (N = 49) 13 to 20 years of age (mean = 16.95). While there were no differences between youths with and without SI in self-reported emotional responses to negative pictures, youths with SI activated the dorsolateral prefrontal cortex more than youths without SI on trials in which they attempted to regulate their emotional responses compared with trials in which they passively viewed negative pictures. In contrast, during passive viewing of negative stimuli, youths with SI activated the dorsolateral prefrontal cortex, temporoparietal junction, and cerebellum less than same-age control subjects. These findings were robust to control subjects for depression and adversity exposure and are consistent with the idea that youths with SI have disrupted emotion regulation, potentially related to differences in recruitment of top-down control regions. In contrast, youths without SI activated regions implicated in emotion regulation even when not directed to effortfully control their emotional response. This is the first study to examine neural function during emotion regulation as a potential neural correlate of risk for SI in adolescents.},
journal = {Biological Psychiatry: Cognitive Neuroscience and Neuroimaging},
author = {Miller, Adam Bryant and McLaughlin, Katie A. and Busso, Daniel S. and Brueck, Stephanie and Peverill, Matthew and Sheridan, Margaret A.},
month = sep,
year = {2017},
keywords = {Adolescent suicide, cognitive reappraisal, DLPFC, emotion regulation, Suicidal ideation, top-down control},
file = {ScienceDirect Full Text PDF:C\:\\Users\\Matt Peverill\\Zotero\\storage\\8UG3VVU3\\Miller et al. - 2017 - Neural Correlates of Emotion Regulation and Adoles.pdf:application/pdf;ScienceDirect Snapshot:C\:\\Users\\Matt Peverill\\Zotero\\storage\\7XSJ4UVI\\S2451902217301489.html:text/html}
}
@article{logue_smaller_2018,
title = {Smaller hippocampal volume in posttraumatic stress disorder: a multisite {ENIGMA}-{PGC} study: subcortical volumetry results from posttraumatic stress disorder consortia},
volume = {83},
shorttitle = {Smaller {Hippocampal} {Volume} in {Posttraumatic} {Stress} {Disorder}},
doi = {10.1016/j.biopsych.2017.09.006},
abstract = {Background Many studies report smaller hippocampal and amygdala volumes in posttraumatic stress disorder (PTSD), but findings have not always been consistent. Here, we present the results of a large-scale neuroimaging consortium study on PTSD conducted by the Psychiatric Genomics Consortium (PGC)–Enhancing Neuroimaging Genetics through Meta-Analysis (ENIGMA) PTSD Working Group. Methods We analyzed neuroimaging and clinical data from 1868 subjects (794 PTSD patients) contributed by 16 cohorts, representing the largest neuroimaging study of PTSD to date. We assessed the volumes of eight subcortical structures (nucleus accumbens, amygdala, caudate, hippocampus, pallidum, putamen, thalamus, and lateral ventricle). We used a standardized image-analysis and quality-control pipeline established by the ENIGMA consortium. Results In a meta-analysis of all samples, we found significantly smaller hippocampi in subjects with current PTSD compared with trauma-exposed control subjects (Cohen's d = −0.17, p =.00054), and smaller amygdalae (d = −0.11, p =.025), although the amygdala finding did not survive a significance level that was Bonferroni corrected for multiple subcortical region comparisons (p {\textless}.0063). Conclusions Our study is not subject to the biases of meta-analyses of published data, and it represents an important milestone in an ongoing collaborative effort to examine the neurobiological underpinnings of PTSD and the brain's response to trauma. © 2017},
number = {3},
journal = {Biological Psychiatry},
author = {Logue, M.W. and van, Rooij and Dennis, E.L. and Davis, S.L. and Hayes, J.P. and Stevens, J.S. and Densmore, M. and Haswell, C.C. and Ipser, J. and Koch, S.B.J. and Korgaonkar, M. and Lebois, L.A.M. and Peverill, M. and Baker, J.T. and Boedhoe, P.S.W. and Frijling, J.L. and Gruber, S.A. and Harpaz-Rotem, I. and Jahanshad, N. and Koopowitz, S. and Levy, I. and Nawijn, L. and O'Connor, L. and Olff, M. and Salat, D.H. and Sheridan, M.A. and Spielberg, J.M. and van, Zuiden and Winternitz, S.R. and Wolff, J.D. and Wolf, E.J. and Wang, X. and Wrocklage, K. and Abdallah, C.G. and Bryant, R.A. and Geuze, E. and Jovanovic, T. and Kaufman, M.L. and King, A.P. and Krystal, J.H. and Lagopoulos, J. and Bennett, M. and Lanius, R. and Liberzon, I. and McGlinchey, R.E. and McLaughlin, K.A. and Milberg, W.P. and Miller, M.W. and Ressler, K.J. and Veltman, D.J. and Stein, D.J. and Thomaes, K. and Thompson, P.M. and Morey, R.A.},
year = {2018},
keywords = {Amygdala, Childhood trauma, gender differences, Hippocampus, PTSD, Structural MRI},
pages = {244--253},
file = {SCOPUS Snapshot:C\:\\Users\\Matt Peverill\\Zotero\\storage\\PSVVMAK6\\display.html:text/html}
}
@article{madhyastha_current_2017,
title = {Current methods and limitations for longitudinal {fMRI} analysis across development},
issn = {1878-9307},
doi = {10.1016/j.dcn.2017.11.006},
abstract = {The human brain is remarkably plastic. The brain changes dramatically across development, with ongoing functional development continuing well into the third decade of life and substantial changes occurring again in older age. Dynamic changes in brain function are thought to underlie the innumerable changes in cognition, emotion, and behavior that occur across development. The brain also changes in response to experience, which raises important questions about how the environment influences the developing brain. Longitudinal functional magnetic resonance imaging (fMRI) studies are an essential means of understanding these developmental changes and their cognitive, emotional, and behavioral correlates. This paper provides an overview of common statistical models of longitudinal change applicable to developmental cognitive neuroscience, and a review of the functionality provided by major software packages for longitudinal fMRI analysis. We demonstrate that there are important developmental questions that cannot be answered using available software. We propose alternative approaches for addressing problems that are commonly faced in modeling developmental change with fMRI data.},
language = {eng},
journal = {Developmental Cognitive Neuroscience},
author = {Madhyastha, Tara and Peverill, Matthew and Koh, Natalie and McCabe, Connor and Flournoy, John and Mills, Kate and King, Kevin and Pfeifer, Jennifer and McLaughlin, Katie A.},
month = nov,
year = {2017},
pmid = {29196032},
keywords = {Developmental change, Functional magnetic resonance imaging (fMRI), General linear model, Longitudinal modeling, Structural Equation Modeling}
}
@article{sheridan_dimensions_2017,
title = {Dimensions of childhood adversity have distinct associations with neural systems underlying executive functioning},
volume = {29},
issn = {1469-2198},
doi = {10.1017/S0954579417001390},
abstract = {Childhood adversity is associated with increased risk for psychopathology. Neurodevelopmental pathways underlying this risk remain poorly understood. A recent conceptual model posits that childhood adversity can be deconstructed into at least two underlying dimensions, deprivation and threat, that are associated with distinct neurocognitive consequences. This model argues that deprivation (i.e., a lack of cognitive stimulation and learning opportunities) is associated with poor executive function (EF), whereas threat is not. We examine this hypothesis in two studies measuring EF at multiple levels: performance on EF tasks, neural recruitment during EF, and problems with EF in daily life. In Study 1, deprivation (low parental education and child neglect) was associated with greater parent-reported problems with EF in adolescents (N = 169; 13-17 years) after adjustment for levels of threat (community violence and abuse), which were unrelated to EF. In Study 2, low parental education was associated with poor working memory (WM) performance and inefficient neural recruitment in the parietal and prefrontal cortex during high WM load among adolescents (N = 51, 13-20 years) after adjusting for abuse, which was unrelated to WM task performance and neural recruitment during WM. These findings constitute strong preliminary evidence for a novel model of the neurodevelopmental consequences of childhood adversity.},
language = {eng},
number = {5},
journal = {Development and Psychopathology},
author = {Sheridan, Margaret A. and Peverill, Matthew and Finn, Amy S. and McLaughlin, Katie A.},
month = dec,
year = {2017},
pmid = {29162183},
pmcid = {PMC5733141},
pages = {1777--1794}
}
@article{rosen_role_2017,
title = {The {Role} of {Visual} {Association} {Cortex} in {Associative} {Memory} {Formation} across {Development}},
issn = {1530-8898},
doi = {10.1162/jocn_a_01202},
abstract = {Associative learning underlies the formation of new episodic memories. Associative memory improves across development, and this age-related improvement is supported by the development of the hippocampus and pFC. Recent work, however, additionally suggests a role for visual association cortex in the formation of associative memories. This study investigated the role of category-preferential visual processing regions in associative memory across development using a paired associate learning task in a sample of 56 youths (age 6-19 years). Participants were asked to bind an emotional face with an object while undergoing fMRI scanning. Outside the scanner, participants completed a memory test. We first investigated age-related changes in neural recruitment and found linear age-related increases in activation in lateral occipital cortex and fusiform gyrus, which are involved in visual processing of objects and faces, respectively. Furthermore, greater activation in these visual processing regions was associated with better subsequent memory for pairs over and above the effect of age and of hippocampal and pFC activation on performance. Recruitment of these visual processing regions mediated the association between age and memory performance, over and above the effects of hippocampal activation. Taken together, these findings extend the existing literature to suggest that greater recruitment of category-preferential visual processing regions during encoding of associative memories is a neural mechanism explaining improved memory across development.},
language = {eng},
journal = {Journal of Cognitive Neuroscience},
author = {Rosen, Maya L. and Sheridan, Margaret A. and Sambrook, Kelly A. and Peverill, Matthew R. and Meltzoff, Andrew N. and McLaughlin, Katie A.},
month = oct,
year = {2017},
pmid = {29064341},
pages = {1--16}
}
@incollection{weisz_harnessing_2017,
title = {Harnessing the {Neuroscience} {Revolution} to {Enhance} {Child} and {Adolescent} {Psychotherapy}},
isbn = {978-1-4625-3005-2},
abstract = {Widely regarded as the standard reference and text on evidence-based therapies--and now substantially revised--this book has introduced tens of thousands of clinicians and students to exemplary treatments for social, emotional, and behavioral problems in children and youth. Concise chapters focus on specific psychotherapy models. The developers of the respective approaches review their conceptual underpinnings, describe how interventions are delivered on a session-by-session basis, and summarize what the research shows about treatment effectiveness. The book explores important questions and challenges facing the field and identifies best practices for treatment dissemination in real-world clinical contexts. ÿ New to This Edition: *Existing chapters significantly revised with the latest treatment research. *Chapters on assessment, personalizing evidence-based therapies, clinical implications of developmental psychopathology, and advances in neuroscience. *Coverage of additional clinical problems (self-injury and suicidal ideation, bulimia nervosa, encopresis) and treatments (Early Start Denver Model for autism spectrum disorder, Internet-assisted and technology-enhanced interventions). *Chapters on implementation in complex mental health systems, as well as international models.ÿ},
language = {en},
booktitle = {Evidence-{Based} {Psychotherapies} for {Children} and {Adolescents}, {Third} {Edition}},
publisher = {Guilford Publications},
author = {Peverill, Matthew and McLaughlin, Katie A.},
editor = {Weisz, John R. and Kazdin, Alan E.},
month = jun,
year = {2017},
note = {Google-Books-ID: cG4RDgAAQBAJ},
keywords = {Medical / Nursing / Psychiatric \& Mental Health, Medical / Psychiatry / Child \& Adolescent, Psychology / Clinical Psychology, Psychology / Psychotherapy / Child \& Adolescent, Social Science / Social Work}
}
@article{lambert_altered_2019,
title = {Altered development of hippocampus-dependent associative learning following early-life adversity},
volume = {38},
issn = {1878-9293},
url = {http://www.sciencedirect.com/science/article/pii/S1878929318303189},
doi = {10.1016/j.dcn.2019.100666},
abstract = {Little is known about how childhood adversity influences the development of learning and memory and underlying neural circuits. We examined whether violence exposure in childhood influenced hippocampus-dependent associative learning and whether differences: a) were broad or specific to threat cues, and b) exhibited developmental variation. Children (n = 59; 8–19 years, 24 violence-exposed) completed an associative learning task with angry, happy, and neutral faces paired with objects during fMRI scanning. Outside the scanner, participants completed an associative memory test for face-object pairings. Violence-exposed children exhibited broad associative memory difficulties that became more pronounced with age, along with reduced recruitment of the hippocampus and atypical recruitment of fronto-parietal regions during encoding. Violence-exposed children also showed selective disruption of associative memory for threat cues regardless of age, along with reduced recruitment of the intraparietal sulcus (IPS) during encoding in the presence of threat. Broad associative learning difficulties may be a functional consequence of the toxic effects of early-life stress on hippocampal and fronto-parietal cortical development. Difficulties in the presence of threat cues may result from enhanced threat processing that disrupts encoding and short-term storage of associative information in the IPS. These associative learning difficulties may contribute to poor life outcomes following childhood violence exposure.},
urldate = {2019-08-23},
journal = {Developmental Cognitive Neuroscience},
author = {Lambert, Hilary K. and Peverill, Matthew and Sambrook, Kelly A. and Rosen, Maya L. and Sheridan, Margaret A. and McLaughlin, Katie A.},
month = aug,
year = {2019},
keywords = {Associative learning, Childhood adversity, Early-life stress, Hippocampus, Violence},
pages = {100666},
file = {ScienceDirect Full Text PDF:C\:\\Users\\Matt Peverill\\Zotero\\storage\\W76QIAZZ\\Lambert et al. - 2019 - Altered development of hippocampus-dependent assoc.pdf:application/pdf;ScienceDirect Snapshot:C\:\\Users\\Matt Peverill\\Zotero\\storage\\3JHNENBN\\S1878929318303189.html:text/html}
}
@article{peverill_atypical_2019,
title = {Atypical {Prefrontal}–{Amygdala} {Circuitry} {Following} {Childhood} {Exposure} to {Abuse}: {Links} {With} {Adolescent} {Psychopathology}},
issn = {1077-5595},
shorttitle = {Atypical {Prefrontal}–{Amygdala} {Circuitry} {Following} {Childhood} {Exposure} to {Abuse}},
url = {https://doi.org/10.1177/1077559519852676},
doi = {10.1177/1077559519852676},
abstract = {Adverse childhood experiences have been associated with more negative coupling between the ventromedial prefrontal cortex (vmPFC) and amygdala, a brain network involved in emotion regulation in both children and adults. This pattern may be particularly likely to emerge in individuals exposed to threatening experiences during childhood (e.g., exposure to child abuse), although this has not been examined in prior research. We collected functional magnetic resonance imaging data on 57 adolescents during an emotion regulation task. Greater negative functional connectivity between vmPFC and amygdala occurred during viewing of negative compared to neutral images. This vmPFC-amygdala task-related functional connectivity was more negative in adolescents exposed to physical, sexual, or emotional abuse than those without a history of maltreatment and was associated with abuse severity. This pattern of more negative functional connectivity was associated with higher levels of externalizing psychopathology concurrently and 2 years later. Greater negative connectivity in the vmPFC-amygdala network during passive viewing of negative images may reflect disengagement of regulatory responses from vmPFC in situations eliciting strong amygdala reactivity, potentially due to stronger appraisals of threat in children exposed to early threatening environments. This pattern may be adaptive in the short term but place adolescents at higher risk of psychopathology later in life.},
language = {en},
urldate = {2019-08-23},
journal = {Child Maltreatment},
author = {Peverill, Matthew and Sheridan, Margaret A. and Busso, Daniel S. and McLaughlin, Katie A.},
month = may,
year = {2019},
pages = {1077559519852676},
file = {SAGE PDF Full Text:C\:\\Users\\Matt Peverill\\Zotero\\storage\\UUGSMZH2\\Peverill et al. - 2019 - Atypical Prefrontal–Amygdala Circuitry Following C.pdf:application/pdf}
}
@article{jenness_dynamic_2019,
title = {Dynamic associations between stressful life events and adolescent internalizing psychopathology in a multiwave longitudinal study},
volume = {128},
issn = {0021-843X},
url = {http://offcampus.lib.washington.edu/login?url=http://search.ebscohost.com/login.aspx?direct=true&db=pdh&AN=2019-43757-011&site=ehost-live},
doi = {10.1037/abn0000450},
abstract = {Associations between stressful life events (SLEs) and internalizing psychopathology are complex and bidirectional, involving interactions among stressors across development to predict psychopathology (i.e., stress sensitization) and psychopathology predicting greater exposure to SLEs (i.e., stress generation). Although stress sensitization and generation theoretical models inherently focus on within-person effects, most previous research has compared average levels of stress and psychopathology across individuals in a sample (i.e., between-person effects). The present study addressed this gap by investigating stress sensitization and stress generation effects in a multiwave, prospective study of SLEs and adolescent depression and anxiety symptoms. Depression, anxiety, and SLE exposure were assessed every 3 months for 2 years (8 waves of data) in a sample of adolescents (n = 382, aged 11 to 15 at baseline). Multilevel modeling revealed within-person stress sensitization effects such that the association between within-person increases in SLEs and depression, but not anxiety, symptoms were stronger among adolescents who experienced higher average levels of SLEs across 2 years. We also observed within-person stress generation effects, such that adolescents reported a greater number of dependent-interpersonal SLEs during time periods after experiencing higher levels of depression at the previous wave than was typical for them. Although no within-person stress generation effects emerged for anxiety, higher overall levels of anxiety predicted greater exposure to dependent-interpersonal SLEs. Our findings extend prior work by demonstrating stress sensitization in predicting depression following normative forms of SLEs and stress generation effects for both depression and anxiety using a multilevel modeling approach. Clinical implications include an individualized approach to interventions. (PsycINFO Database Record (c) 2019 APA, all rights reserved)},
number = {6},
urldate = {2019-08-23},
journal = {Journal of Abnormal Psychology},
author = {Jenness, Jessica L. and Peverill, Matthew and King, Kevin M. and Hankin, Benjamin L. and McLaughlin, Katie A.},
month = aug,
year = {2019},
keywords = {Adolescent Psychopathology, adolescents, anxiety, Anxiety, depression, Life Experiences, Major Depression, Psychopathology, Sensitization, Stress, stress generation, stress sensitization},
pages = {596--609},
file = {EBSCO Full Text:C\:\\Users\\Matt Peverill\\Zotero\\storage\\UWRTUHYF\\Jenness et al. - 2019 - Dynamic associations between stressful life events.pdf:application/pdf}
}
@article{dennis_altered_2019,
title = {Altered {White} {Matter} {Microstructural} {Organization} in {Post}-{Traumatic} {Stress} {Disorder} across 3,049 {Adults}: {Results} from the {PGC}-{ENIGMA} {PTSD} {Consortium}},
copyright = {© 2019, Posted by Cold Spring Harbor Laboratory. This pre-print is available under a Creative Commons License (Attribution-NonCommercial-NoDerivs 4.0 International), CC BY-NC-ND 4.0, as described at http://creativecommons.org/licenses/by-nc-nd/4.0/},
shorttitle = {Altered {White} {Matter} {Microstructural} {Organization} in {Post}-{Traumatic} {Stress} {Disorder} across 3,049 {Adults}},
url = {https://www.biorxiv.org/content/10.1101/677153v1},
doi = {10.1101/677153},
abstract = {{\textless}h3{\textgreater}Abstract{\textless}/h3{\textgreater} {\textless}p{\textgreater}A growing number of studies have examined alterations in white matter organization in people with posttraumatic stress disorder (PTSD) using diffusion MRI (dMRI), but the results have been mixed, which may be partially due to relatively small sample sizes among studies. Altered structural connectivity may be both a neurobiological vulnerability for, and a result of, PTSD. In an effort to find reliable effects, we present a multi-cohort analysis of dMRI metrics across 3,049 individuals from 28 cohorts currently participating in the PGC-ENIGMA PTSD working group (a joint partnership between the Psychiatric Genomics Consortium and the Enhancing NeuroImaging Genetics through Meta-Analysis consortium). Comparing regional white matter metrics across the full brain in 1,446 individuals with PTSD and 1,603 controls (2152 males/897 females) between ages 18-83, 92\% of whom were trauma-exposed, we report associations between PTSD and disrupted white matter organization measured by lower fractional anisotropy (FA) in the tapetum region of the corpus callosum (Cohen’s \textit{d}=−0.12, \textit{p}=0.0021). The tapetum connects the left and right hippocampus, structures for which structure and function have been consistently implicated in PTSD. Results remained significant/similar after accounting for the effects of multiple potentially confounding variables: childhood trauma exposure, comorbid depression, history of traumatic brain injury, current alcohol abuse or dependence, and current use of psychotropic medications. Our results show that PTSD may be associated with alterations in the broader hippocampal network.{\textless}/p{\textgreater}},
language = {en},
urldate = {2019-08-23},
journal = {bioRxiv},
author = {Dennis, Emily L. and Disner, Seth G. and Fani, Negar and Salminen, Lauren E. and Logue, Mark and Clarke, Emily K. and Haswell, Courtney C. and Averill, Christopher L. and Baugh, Lee A. and Bomyea, Jessica and Bruce, Steven E. and Cha, Jiook and Choi, Kyle and Davenport, Nicholas D. and Densmore, Maria and Plessis, Stefan du and Forster, Gina L. and Frijling, Jessie L. and Gönenc, Atilla and Gruber, Staci and Grupe, Daniel W. and Guenette, Jeffrey P. and Hayes, Jasmeet and Hofmann, David and Ipser, Jonathan and Jovanovic, Tanja and Kelly, Sinead and Kennis, Mitzy and Kinzel, Philipp and Koch, Saskia BJ and Koerte, Inga and Koopowitz, Sheri and Korgaonkar, Mayuresh and Krystal, John and Lebois, Lauren AM and Li, Gen and Magnotta, Vincent A. and Manthey, Antje and May, Geoffrey J. and Menefee, Deleene S. and Nawijn, Laura and Nelson, Steven M. and Neufeld, Richard WJ and Nitschke, Jack B. and O’Doherty, Daniel and Peverill, Matthew and Ressler, Kerry and Roos, Annerine and Sheridan, Margaret A. and Sierk, Anika and Simmons, Alan and Simons, Raluca M. and Simons, Jeffrey S. and Stevens, Jennifer and Suarez-Jimenez, Benjamin and Sullivan, Danielle R. and Théberge, Jean and Tran, Jana K. and Heuvel, Leigh van den and Werff, Steven JA van der and Rooij, Sanne JH van and Zuiden, Mirjam van and Velez, Carmen and Verfaellie, Mieke and Vermeiren, Robert RJM and Wade, Benjamin SC and Wager, Tor and Walter, Henrik and Winternitz, Sherry and Wolff, Jonathan and York, Gerald and Zhu, Ye and Zhu, Xi and Abdallah, Chadi G. and Bryant, Richard and Daniels, Judith K. and Davidson, Richard J. and Fercho, Kelene A. and Franz, Carol and Geuze, Elbert and Gordon, Evan M. and Kaufman, Milissa L. and Kremen, William and Lagopoulos, Jim and Lanius, Ruth A. and Lyons, Michael J. and McCauley, Stephen R. and McGlinchey, Regina and McLaughlin, Katie A. and Milberg, William and Neria, Yuval and Olff, Miranda and Seedat, Soraya and Shenton, Martha and Sponheim, Scott R. and Stein, Dan J. and Stein, Murray B. and Straube, Thomas and Tate, David F. and Wee, Nic JA van der and Veltman, Dick J. and Wang, Li and Wilde, Elisabeth A. and Thompson, Paul M. and Kochunov, Peter and Jahanshad, Neda and Morey, Rajendra A.},
month = jun,
year = {2019},
pages = {677153},
file = {Full Text PDF:C\:\\Users\\Matt Peverill\\Zotero\\storage\\QZPA9BNW\\Dennis et al. - 2019 - Altered White Matter Microstructural Organization .pdf:application/pdf;Snapshot:C\:\\Users\\Matt Peverill\\Zotero\\storage\\F36J8K9X\\677153v1.html:text/html}
}